David Albertini Biography
Yesterday we posted an announcement of David Albertini joining the Bedford Research Foundation as Professor and Chair of the Department of Developmental Cell Biology.
David Albertini received his Ph.D. in 1975 from Harvard University working on the cell biology of the mammalian ovary. After postdoctoral work at the University of Connecticut Health Center, he returned to Harvard Medical School as an Assistant Professor of Anatomy and Cell Biology from 1977-1984, and was Professor at Tufts University Schools of Medicine until 2004. At Tufts, he served as Chair of the Department of Anatomy and Cell Biology (1996-2000), Director of the Center for Reproduction (1999-2003) and Director of the Confocal Microscopy core (1988-1999). From 2004-2016, he was Hall Professor of Molecular Medicine at the Kansas University Medical Center. In 2016 he became the Director of Laboratories and Senior Scientist at the Center for Human Reproduction in New York City. He serves as the Editor-in-Chief of the Journal of Assisted Reproduction and Genetics, an official journal of the American Society for Reproductive Medicine (2009-present). In 2020, David joined the Bedford Research Foundation team where he continues his research interests in reproductive medicine and biology. He is the recipient of many awards including a Basil O’Connor fellowship from the March of Dimes, the Hammond Medal from the Society for Reproduction and Fertility (UK), and the Founder’s Lecturer for the Australian Society of Reproductive Biology, The Beacon Award FIR course MBL, and recently was recognized as an Honorary Member of the International Society for In Vitro Fertilization (ISIVF). He is presently a Visiting Scientist at The Rockefeller University and Center for Human Reproduction, continues to serve on scientific advisory boards, and is a frequent invited speaker here and abroad. His most recent research at the Bedford Research Foundation is aimed at developing clinically useful stem cells from activated human eggs and understanding the mechanisms underlying ovarian and oocyte aging in women.